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What Causes Systemic Sclerosis? You Can Blame the Children or the Cytomegalovirus or Perhaps Both
CME credit is no longer available for this conference.
Peter Barland, M.D.

Dr. Barland reports no commercial conflict of interest.


Release Date: 06/03/1998
Termination Date: 06/03/2001

Estimated time to complete: 1 hour(s).

Albert Einstein College of Medicine designates this enduring material for a maximum of 1 AMA PRA Category 1 Credit(s)™. Physicians should claim only the credit commensurate with the extent of their participation in the activity.

Albert Einstein College of Medicine is accredited by the Accreditation Council for Continuing Medical Education to provide continuing medical education for physicians.
 
Learning Objectives
Upon completion of this Cyberounds®, you should be able to:
  • Discuss the major clinical manifestations of systemic sclerosis arising from increased collagen production and from arterial vasculopathy
  • Describe the recent evidence for the persistence of fetal cells in parous women with systemic sclerosis and how these cells might produce a chronic graft versus host condition resembling systemic sclerosis
  • Discuss the scientific rationale for implicating latent human cytomegalovirus infection in the pathogenesis of systemic sclerosis.

 
This conference may include discussion of commercial products and services.

The opinions expressed herein are those of the authors and do not necessarily represent the views of the sponsor or its publisher. Please review complete prescribing information of specific drugs or combination of drugs, including indications, contraindications, warnings and adverse effects before administering pharmacologic therapy to patients.
 
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